Gesunde Fette: Endnoten (Hinweise im Text) Dr. Joseph Mercola: Der optimale Kraftstoff für ihren Körper. Mit der Mitochondrien Therapie gesund und fit bis ins hohe Alter.

Verzeichnis

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Nüsse + Samen
Danksagungen

Einleitung

  • (001) K. M. Adams, W. S. Butsch und M. Kohlmeier, »The State of Nutrition Education at US Medical Schools«, Journal of Biomedical Education, Jg. 2015 (Januar 2015), Article ID 357627, 7 Seiten. DOI:10.1155/2015/357627.
  • (002) »Cancer Facts & Figures 2016«, American Cancer Society, Atlanta, Georgia, 2016, http://www.cancer.org/acs/groups/content/@research/documents/document/acspc-047079.pdf, aufgerufen am 14. August 2017.
  • (003) »Global Cancer Facts & Figures«, 3. Ausgabe, American Cancer Society, Atlanta, Georgia, 2015, http://www.cancer.org/acs/groups/content/@research/documents/document/acspc-044738.pdf, aufgerufen am 14. August 2017.
  • (004) N. Howlader et al. (Hrsg.), »SEER Cancer Statistics Review, 1975–2013«, National Cancer Institute, Bethesda, MD, April 2016, http://seer.cancer.gov/csr/1975_2013/, aufgerufen am 14. August 2017.
  • (005) Den in der Originalausgabe Fat for Fuel vewendeten Begriff Mitochondrial Metabolic Therapy (MMT) benennen wir in der vorliegenden deutschen Fassung mit der Bezeichnung Mitochondrien-Therapie. Die Abkürzung MMT wird beibehalten. Sie bezieht sich auf die englische Originalbezeichnung. [Anmerkung der Redaktion]
  • (006) M. Harper, »David Graham on the Vioxx Verdict«, Forbes.com, 19. August 2005, http://www.forbes.com/2005/08/19/merck-vioxx-graham_cx_mh_0819graham.html, aufgerufen am 14. August 2017.

Kapitel 1

  • (007) N. Lane, Power, Sex, Suicide: Mitochondria and the Meaning of Life. Oxford University Press, New York 2006, S. 3.
  • (008) Ebd.
  • (009) Ebd., Pos. 5926.
  • (010) »Our Best Days Are Yours«, Kellogg᾽s, https://www.kelloggs.com/en_US/who-we-are/our-history.html, aufgerufen am 2. Dezember 2016.
  • (011) L. B. Wrenn, Cinderella of the New South. University of Tennessee Press, Knoxville, TN 1995, S. 84.
  • (012) T. G. Graham und D. Ramsey, The Happiness Diet. Rodale Books, New York 2012, S. 25.
  • (013) F. G. Mather, »Waste Products: Cotton-Seed Oil«, Popular Science Monthly, Mai 1894, S. 104.
  • (014) Graham und Ramsey, The Happiness Diet. A.a. O.
  • (015) »Our Heritage«, Crisco, http://www.crisco.com/about_crisco/history.aspx, aufgerufen am 14. August 2017.
  • (016) S. Gokhale, »Marketing Crisco«, Weston A. Price Foundation, 25. Juni 2013, http://www.westonaprice.org/health-topics/marketing-crisco/, aufgerufen am 14. August 2017.
  • (017) Graham und Ramsey, The Happiness Diet. A.a. O.
  • (018) L. Blasbalg et al., »Changes in Consumption of Omega-3 and Omega-6 Fatty Acids in the United States During the 20th Century«, American Journal of Clinical Nutrition, 93, Nr. 5 (Mai 2011): 950–62, DOI: 10.3945/ajcn.110.006643, Epub 2. März 2011.
  • (019) S. F. Halabi, Food and Drug Regulation in an Era of Globalized Markets. Academic Press, Cambridge, MA 2015, S. 148.
  • (020) T. Neltner, M. Maffini, »Generally Recognized as Secret: Chemicals Added to Food in the United States«, National Resources Defense Council, April 2014, https://www.nrdc.org/sites/default/files/safety-loophole-for-chemicals-in-food-report.pdf, aufgerufen am 14. August 2017.
  • (021) R. J. de Souza et al., »Intake of Saturated and Trans Unsaturated Fatty Acids and Risk of All Cause Mortality, Cardiovascular Disease, and Type 2 Diabetes: Systematic Review and Meta-analysis of Observational Studies«, BMJ (2015): 351, DOI: 10.1136/bmj. h3978.
  • (022) V. T. Samuel, K. F. Petersen und G. I. Shulman, »Lipid-induced Insulin Resistance: Unraveling the Mechanism«, Lancet, 375, (2010): 2267–77, DOI: 10.1016/S0140-6736(10)60408-4.
  • (023) K. Kavanagh et al., »Trans Fat Diet Induces Abdominal Obesity and Changes in Insulin Sensitivity in Monkeys«, Obesity, 15, Nr. 7 (Juli 2007): 1675–84, DOI: 10.1038/oby.2007.200.
  • (024) M. C. Morris et al., »Dietary fats and the risk of incident Alzheimer᾽s disease«, Archives of Neurology, 60, Nr. 2 (2003): 194–200, DOI: 10.1001 /archneur.60.2.194.
  • (025) C. M. Benbrook, »Impacts of Genetically Engineered Crops on Pesticide Use in the U.S. – the First Sixteen Years«, Environmental Sciences Europe, 24, Nr. 1 (2012): 24, DOI: 10.1186/2190-4715-24-24.
  • (026) N. Defarge et al., »Co-Formulants in Glyphosate-Based Herbicides Disrupt Aromatase Activity in Human Cells below Toxic Levels«, International Journal of Environmental Research and Public Health, 13, Nr. 3 (2016): 264, DOI: 10.3390/ijerph13030264.
  • (027) A. Keys, »Mediterranean Diet and Public Health: Personal Reflections«, American Journal of Clinical Nutrition, 61, Nr. 6 Suppl. (1995): 1321S–1323S.
  • (028) A. Keys, »Atherosclerosis: A Problem in Newer Public Health«, Journal of Mt. Sinai Hospital, New York, 20, Nr. 2 (Juli–August 1953): 134.
  • (029) N. Teichholz, The Big Fat Surprise, Simon & Schuster, New York 2014, S. 32–33.
  • (030) Central Committee for Medical and Community Program of the American Heart Association, »Dietary Fat and Its Relation to Heart Attacks and Strokes«, Circulation, 23 (1961): 133–36. http://circ.ahajournals.org/content/circulationaha/23/1/133.full.pdf, aufgerufen am 14. August 2017.
  • (031) H. M. Marvin, 1924–1964: The 40 Year War on Heart Disease, American Heart Association, New York 1964.
  • (032) A. Keys, »Coronary Heart Disease in Seven Countries«, Circulation, 41, Nr. 1 (1970): 1186–95.
  • (033) Dietary Guidelines Advisory Committee, »History of the Dietary Guidelines for Americans«, Nutrition and Health: Dietary Guidelines for Americans, 2005, U.S. Department of Health and Human Services, https://health.gov/dietaryguidelines/dga2005/report/html/G5_History.htm, aufgerufen am 14. August
  • (034) Z. Harcombe et al., »Evidence from Randomised Controlled Trials Did Not Support the Introduction of Dietary Fat Guidelines in 1977 and 1983: A Systematic Review and Metaanalysis«, Open Heart, 2, Nr. 1 (2015), DOI: 10.1136/openhrt-2014-000196.
  • (035) U.S. Department of Health and Human Services und U.S. Department of Agriculture, »Key Recommendations: Components of Healthy Eating Patterns«, 2015–2020 Dietary Guidelines for Americans, 8. Ausgabe (Dezember 2015): 15, https://health.gov/dietaryguidelines/2015/guidelines/chapter-1/key-recommendations/#footnote-4, aufgerufen am 14. August 2017.
  • (036) Centers for Disease Control and Prevention, Division of Diabetes Translation, »Long-term Trends in Diabetes«, (2016), https://www.cdc.gov/diabetes/statistics/slides/long_term_trends.pdf, aufgerufen am 2. Oktober 2017.
  • (037) C. D. Fryar, M. Carroll und C. Ogden, Division of Health and Nutrition Examination Surveys, »Prevalence of Overweight, Obesity, and Extreme Obesity Among Adults Aged 20 and Over: United States, 1960–1962 Through 2013–2014«, Tabelle 1, Centers for Disease Control and Prevention, http://www.cdc.gov/nchs/data/hestat/obesity_adult_13_14/obesity_adult_13_14.htm#Figure, aufgerufen am 14. August 2017.
  • (038) N. Howlader et al. (Hrsg.), »SEER Cancer Statistics Review, 1975–2013«, a. a.O.
  • (039) »SEER Stat Fact Sheets: Cancer of Any Site«, National Cancer Institute, http://seer.cancer.gov/statfacts/html/all.html, aufgerufen am 14. August 2017.
  • (040) A. Heidenreich et al., »Forecasting the Future of Cardiovascular Disease in the United States«, Circulation, 123, Nr. 8 (2011): 933–944, DOI: 10.1161/CIR.0b013e31820a55f5.
  • (041) P. Leren, »The Effect of Plasma-Cholesterol-Lowering Diet in Male Survivors of Myocardial Infarction: A Controlled Clinical Trial«, Bulletin of the New York Academy of Medicine, 44, Nr. 8 (1968):S. 1012–20.
  • (042) S. Dayton et al., »A Controlled Clinical Trial of a Diet High in Unsaturated Fat in Preventing Complications of Atherosclerosis«, Circulation, 40 (1969): II-1–II-63, DOI: 10.1161/01. CIR.40.1S2. II-1.
  • (043) I. D. Frantz et al., »Test of effect of lipid lowering by diet on cardiovascular risk. The Minnesota Coronary Survey«, Arteriosclerosis, 9, Nr. 1, (Januar–Februar 1989): 129–35, DOI: 10.1161/01. ATV.9.1.129.
  • (044) O. Turpeinen et al., »Dietary Prevention of Coronary Heart Disease: The Finnish Mental Hospital Study«, International Journal of Epidemiology, 9, Nr. 2 (1979): 99–118, DOI: 10.1093/ije/8.2.99.
  • (045) »Controlled Trial of Soya-Bean Oil in Myocardial Infarction«, The Lancet, 292, Nr. 7570 (1968): 693–700, DOI: 10.1016/S0140-6736(68)90746-0.
  • (046) Multiple Risk Factor Intervention Trial Group: »Public Annual Report, Multiple Risk Factor Intervention Trial, June 30, 1975 to July 1, 1976«, Journal of the American Medical Association, 248, Nr. 12 (1982): 1465–77, https://clinicaltrials.gov/ct2/show/NCT00000487, aufgerufen am 14. August. 2017.
  • (047) P. W. Siri-Tarino et al., »Meta-analysis of Prospective Cohort Studies Evaluating the Association of Saturated Fat with Cardiovascular Disease«, American Journal of Clinical Nutrition, 91, Nr. 3 (2010):  535–46, DOI:10.3945/ajcn.2009.27725.
  • (048) R. U. Chowdhury et al., »Association of Dietary, Circulating, and Supplement Fatty Acids With Coronary Risk: A Systematic Review and Meta-analysis«, Annals of Internal Medicine, 160 (2014): 398–406, DOI: 10.7326/M13-1788.
  • (049) De Souza et al., »Intake of Saturated and Trans Unsaturated Fatty Acids and Risk of All Cause Mortality, Cardiovascular Disease, and Type 2 Diabetes.«, a. a.O.
  • (050) C. E. Ramsden et al., »Use of Dietary Linoleic Acid for Secondary Prevention of Coronary Heart Disease and Death: Evaluation of Recovered Data From the Sydney Diet Heart Study and Updated Meta-analysis,« BMJ, 346 (2013), DOI: 0.1136/bmj. e8707.
  • (051) Ebd.
  • (052) M. A. Austin et al., »Low-Density Lipoprotein Subclass Patterns and Risk of Myocardial Infarction«, Journal of the American Medical Association, 260, Nr. 13 (1988): 1917–21, DOI: 10.1001/jama.1988.03410130125037.
  • (053) D. M. Dreon et al., »Change in Dietary Saturated Fat Intake Is Correlated with Change in Mass of Large Low-Density-Lipoprotein Particles in Men«, American Journal of Clinical Nutrition, 67, Nr. 5 (1998): 828–36. 48.
  • (054) K. Gunnars, »Saturated Fat, Good or Bad?« Authority Nutrition, https://authoritynutrition.com/saturated-fat-good-or-bad/, aufgerufen am 14. August 2017.
  • (055) P. W. Siri-Tarino et al., »Saturated Fat, Carbohydrate, and Cardiovascular Disease«, American Journal of Clinical Nutrition, 91, Nr. 3 (2010): 502–9, DOI: 10.3945/ajcn.2008.26285.

Kapitel 2

  • (056) L. Cordain, »The Nutritional Characteristics of a Contemporary Diet Based Upon Paleolithic Food Groups«, Journal of the American Nutraceutical Association, 5, Nr. 5, (2002): 15–24.
  • (057) J. J. Meidenbauer, P. Mukherjee und T. N. Seyfried, »The Glucose Ketone Index Calculator: A Simple Tool to Monitor Therapeutic Efficacy for Metabolic Management of Brain Cancer«, Nutrition & Metabolism, 12 (2015): 12, DOI: 10.1186/s12986-015-0009-2.
  • (058) R. Agrawal und F. Gomez-Pinilla, »›Metabolic Syndrome‹ in the Brain: Deficiency in Omega-3 Fatty Acid Exacerbates Dysfunctions in Insulin Receptor Signalling and Cognition«, The Journal of Physiology, 590, Nr. 10, (2012): 2485, DOI: 10.1113/jphysiol.2012.230078.
  • (059) J. R. Ifland et al., »Refined Food Addiction: A Classic Substance Use Disorder«, Medical Hypotheses, 72, Nr. 5, (Mai 2009): 518–26, DOI: 10.1016/j. mehy.2008.11.035.
  • (060) T. R. Nansel et al., »Greater Food Reward Sensitivity Is Associated with More Frequent Intake of Discretionary Foods in a Nationally Representative Sample of Young Adults«, Frontiers in Nutrition, 3, Nr. 33, 18. August 2016, DOI: 10.3389/fnut.2016.00033.
  • (061) S. D. Phinney und J. S. Volek, The Art and Science of Low-Carbohydrate Living. Beyond Obesity LLC, Miami, FL 2011, S. 10.
  • (062) G. D. Maurer et al., »Differential Utilization of Ketone Bodies by Neurons and Glioma Cell Lines: a Rationale for Ketogenic Diet as Experimental Glioma Therapy«, BMC Cancer, 11 (2011): 315, DOI: 10.1186/1471-2407-11-315.
  • (063) R. Sender, S. Fuchs und R. Milo, »Revised Estimates for the Number of Human and Bacteria Cells in the Body«, PLoS Biology, 14, Nr. 8 (2016): e1002533, DOI: 10.1371/journal. pbio.1002533.
  • (064) R. Rosedale, »Life, Death, Food and the Disease of Aging«, Vortrag an der American Academy of Anti-Aging in Orlando, Florida, 2011.
  • (065) C. E. Forsythe et al., »Comparison of Low Fat and Low Carbohydrate Diets on Circulation Fatty Acid Composition and Markers of Inflammation«, Lipids, 43, Nr. 1 (2008): 65–77, DOI: 10.1007/s11745-007-3132-7.
  • (066) S. McKenzie, »Yoshinori Ohsumi Wins Nobel Prize for Medical Research on Cells«, CNN.com, 3. Oktober 2016, http://www.cnn.com/2016/10/03/health/nobel-prize-2016-physiology-medicine-yoshinori-ohsumi/, aufgerufen am 14. August 2017.
  • (067) K. J. Bough et al., »Mitochondrial Biogenesis in the Anticonvulsant Mechanism of the Ketogenic Diet«, Annals of Neurology, 60 (2006): 223–35, DOI: 10.1002/ana.20899.
  • (068) P. J. Cox, K. Clarke, »Acute Nutritional Ketosis: Implications for Exercise Performance and Metabolism«, Extreme Physiology & Medicine, 3 (2014): 1, DOI: 10.1186/2046-7648-3-17.
  • (069) O. E. Owen et al., »Liver and Kidney Metabolism During Prolonged Starvation«, Journal of Clinical Investigation, 48, Nr. 3 (1969): 574–83.
  • (070) M. Akram, »A Focused Review of the Role of Ketone Bodies in Health and Disease«, Journalof Medicinal Food, 16, Nr. 11 (November 2013): 965–67, DOI: 10.1089/jmf.2012.2592.
  • (071) Ebd.
  • (072) Phinney und Volek, The Art and Science of Low-Carbohydrate Living, S. 10, A.a. O.
  • (073) Interview mit Dr. Jeff Volek, http://articles.mercola.com/sites/articles/archive/2016/01/31/high-fat-low-carb-diet-benefits.aspx, aufgerufen am 14. August 2017.
  • (074) J. C. Newman und E. Verdin, »ß-hydroxybutyrate: Much More Than a Metabolite«, Diabetes Research and Clinical Practice, 106, Nr. 2 (2014): 173–81, DOI: 10.1016/j. diabres.2014.08.009.
  • (075) A. Paoli et al., »Ketogenic Diet in Neuromuscular and Neurodegenerative Diseases«, BioMed Research International, 2014 (2014), DOI:10.1155/2014/474296.
  • (076) M. A. McNally und A. L. Hartman, »Ketone Bodies in Epilepsy«, Journal of Neurochemistry, 121, Nr. 1 (2012): 28–35, DOI: 10.1111/j.1471-4159.2012.07670. x.
  • (077) J. Moore, Keto Clarity. Victory Belt Publishing, Las Vegas, NV 2014, S. 58.
  • (078) A. J. Brown, »Low-Carb Diets, Fasting and Euphoria: Is There a Link between Ketosis and Gamma-hydroxybutyrate (GHB)?«, Medical Hypotheses, 68, Nr. 2 (2007): 268–71, DOI: 10.1016/j. mehy.2006.07.043.

Kapitel 3

  • (079) E. L. Knight et al., »The Impact of Protein Intake on Renal Function Decline in Women with Normal Renal Function or Mild Renal Insufficiency«, Annals of Internal Medicine, 138, Nr. 6 (2003): 460–67, DOI: 10.7326/0003-4819-138-6-200303180-00009.
  • (080) M. I. Frisard et al., »Effect of 6-Month Calorie Restriction on Biomarkers of Longevity, Metabolic Adaptation, and Oxidative Stress in Overweight Individuals: A Randomized Controlled Trial«, http://jamanetwork.com/journals/jama/fullarticle/1108368, aufgerufen am 14. August 2017.
  • (081) M. E. Levine et al., »Low Protein Intake Is Associated with a Major Reduction in IGF-1, Cancer, and Overall Mortality in the 65 and Younger but Not Older Population«, Cell Metabolism, 19, Nr. 3 (2014): 407–17, DOI: 10.1016/j. cmet.2014.02.006.
  • (082) J. Guevara-Aguirre et al., »Growth Hormone Receptor Deficiency Is Associated With a Major Reduction in Pro-aging Signaling, Cancer and Diabetes in Humans«, Science Translational Medicine, 3, Nr. 70 (2011): 70, DOI: 10.1126/scitranslmed.3001845.
  • (083) S. I. A. Apelo und D. W. Lamming, »Rapamycin: An InhibiTOR of Aging Emerges From the Soil of Easter Island«, Journal of Gerontology, S. 71, Nr. 7 (2016): 841–849, DOI: 10.1093/gerona/glw090.
  • (084) S. M. Solon-Biet et al., »The Ratio of Macronutrients, Not Caloric Intake, Dictates Cardiometabolic Health, Aging, and Longevity in Ad Libitum-Fed Mice«, Cell Metabolism, S. 19, Nr. 3 (2014): 418–30, DOI: 10.1016/j. cmet.2014.02.009.

Kapitel 4

  • (085) »Ferritin: The Test«, American Association for Clinical Chemistry, https://labtestsonline.org/understanding/analytes/ferritin/tab/test/, aufgerufen am 14. August 2017.
  • (086) E. D. Weinberg, »The Hazards of Iron Loading«, Metallomics, 2, Nr. 11 (November, 2010): S 732–40, DOI: 10.1039/c0mt00023j.
  • (087) M. D. Beaton und P. C. Adams, »Treatment of Hyperferritinemia«, Annals of Hepatology, 11, Nr. 3 (2012): 294–300, PMID: 22481446.
  • (088) G. Ortiz-Estrada et al., »Iron-Saturated Lactoferrin and Pathogenic Protozoa: Could This Protein Be an Iron Source for Their Parasitic Style of Life?« Future Microbiology, 7, Nr. 1 (2012): 149–64, DOI: 10.2217/fmb.11.140.
  • (089) D. J. Fleming et al., »Dietary Factors Associated with the Risk of High Iron Stores in the Elderly Framingham Heart Study Cohort«, American Journal of Clinical Nutrition, 76, Nr. 6 (2002): S. 1375–84, PMID: 12450906.
  • (090) T. Iwasaki et al., »Serum Ferritin Is Associated with Visceral Fat Area and Subcutaneous Fat Area«, Diabetes Care, 28, Nr. 10 (2005): 2486–91, PMID: 16186284.
  • (091) S. K. Park et al., »Association between Serum Ferritin Levels and the Incidence of Obesity in Korean Men: A Prospective Cohort Study«, Endocrine Journal, 61, Nr. 3 (2014): 215–24, DOI: 10.1507/endocrj. EJ13-0173.
  • (092) Ebd.
  • (093) J. M. Fernandez-Real et al., »Serum Ferritin as a Component of the Insulin Resistance Syndrome«, Diabetes Care, 21, Nr. 1 (1998): 62–68, DOI: 10.2337/diacare.21.1.62.
  • (094) J. Montonen et al., »Body Iron Stores and Risk of Type 2 Diabetes: Results from the European Prospective Investigation into Cancer and Nutrition (EPIC)-Potsdam Study«, Diabetologia, 55, Nr. 10 (2012): 2613–21, DOI: 10.1007/s00125-012-2633-y.
  • (095) J. M. Fernandez-Real, A. Lopez-Bermejo und W. Ricart, »Iron Stores, Blood Donation, and Insulin Sensitivity and Secretion«, Clinical Chemistry, 51, Nr. 7 (Juni 2005): 1201–5, DOI: 10.1373/clinchem.2004.046847.
  • (096) B. J. Van Lenten et al., »Lipid-Induced Changes in Intracellular Iron Homeostasis in Vitro and in Vivo«, Journal of Clinical Investigation, 95, Nr. 5 (1995): 2104–10, DOI: 10.1172/JCI117898.
  • (097) N. Stadler, R. A. Lindner und M. J. Davies, »Direct Detection and Quantification of Transition Metal Ions in Human Atherosclerotic Plaques: Evidence for the Presence of Elevated Levels of Iron and Copper«, Arteriosclerosis, Thrombosis, and Vascular Biology, 24 (2004): 949–54, DOI: 10.1161/01. ATV.0000124892.90999. cb.
  • (098) W. B. Kannel et al., »Menopause and Risk of Cardiovascular Disease: The Framingham Study«, Annals of Internal Medicine, 85 (1976): 447–52, DOI: 10.7326/0003-4819-85-4-447.
  • (099) M. A. Lovell et al., »Copper, Iron and Zinc in Alzheimer᾽s Disease Senile Plaques«, Journal of the Neurological Sciences, 158, Nr. 1 (11. Juni 1998): 47–52, DOI: 10.1016/S0022-510X(98)00092-6.
  • (100) K. Jellinger et al., »Brain Iron and Ferritin in Parkinson᾽s and Alzheimer᾽s diseases«, Journal of Neural Transmission, 2 (1990): 327, DOI: 10.1007/BF02252926.
  • (101) G. Bartzokis et al., »Brain Ferritin Iron as a Risk Factor for Age at Onset in Neurodegenerative Diseases«, Annals of the New York Academy of Sciences, 1012 (2004): 224–36, DOI: 10.1196/annals.1306.019.
  • (102) S. Ayton et al., »Ferritin Levels in the Cerebrospinal Fluid Predict Alzheimer᾽s Disease Outcomes and Are Regulated by APOE«, Nature Communications, 6 (2015): 6760, DOI: 10.1038/ncomms7760.
  • (103) W. Z. Zhu et al., »Quantitative MR Phase-Corrected Imaging to Investigate Increased Brain Iron Deposition of Patients with Alzheimer᾽s Disease«, Radiology, 253 (2009): 497–504, DOI: 10.1148/radiol.2532082324.
  • (104) A. A. Alkhateeb und J. R. Connor, »The Significance of Ferritin in Cancer: Anti-Oxidation, Inflammation and Tumorigenesis«, Biochimica et Biophysica Acta, 1836, Nr. 2 (Dez. 2013): S.245–54, DOI: 10.1016/j. bbcan.2013.07.002.
  • (105) J. I. Wurzelmann et al., »Iron Intake and the Risk of Colorectal Cancer«, Cancer Epidemiology, Biomarkers and Prevention, 5, Nr. 7 (1. Juli 1996): 503–7. PMID: 8827353.
  • (106) Y. Deugnier, »Iron and Liver Cancer«, Alcohol, 30, Nr. 2 (2003): 145–50.
  • (107) L. R. Zacharski et al., »Decreased Cancer Risk after Iron Reduction in Patients with Peripheral Arterial Disease: Results from a Randomized Trial«, JNCI: Journal of National Cancer Institute, 100, Nr. 14 (2008): 996–1002, DOI: 10.1093/jnci/djn209.
  • (108) L. Valenti et al., »Association between Iron Overload and Osteoporosis in Patients with Hereditary Hemochromatosis«, Osteoporosis International, 20, Nr. 4 (April 2009): 549–55, DOI: 10.1007/s00198-008-0701-4.
  • (109) »Hemochromatosis«, National Institute of Diabetes and Digestive and Kidney Disease (2016), http://www.niddk.nih.gov/health-information/health-topics/liver-disease/hemochromatosis/Pages/facts.aspx, aufgerufen am 14. August 2017.
  • (110) »Welcome«, Iron Disorders Institute (2016) http://www.hemochromatosis.org/#symptoms, aufgerufen am 14. August 2017.
  • (111) »Serum Iron Test«, MedlinePlus Medical Encyclopedia (2016), https://www.nlm.nih.gov/medlineplus/ency/article/003488.htm, aufgerufen am 14. August 2017.
  • (112) »TIBC, UIBC, and Transferrin Test: Iron Binding Capacity; IBC; Serum Iron-Binding Capacity; Siderophilin; Total Iron Binding Capacity; Unsaturated Iron Binding Capacity«, Lab Tests Online (2016), https://labtestsonline.org/understanding/analytes/tibc/tab/test/, aufgerufen am 14. August 2017.
  • (113) L. Zacharski, »Ferrotoxic Disease: The Next Great Public Health Challenge«, Clinical Chemistry, 60, Nr. 11 (November 2014): 1362–4, DOI: 10.1373/clinchem.2014.231266.
  • (114) P. Mangan, Dumping Iron: How to Ditch This Secret Killer and Reclaim Your Health. Phalanx Press, 2016, Pos. 308–12.
  • (115) Ebd., Pos. 1353–56.
  • (116) Ebd., Pos. 1609–12.
  • (117) Ebd., Pos. 416–18.
  • (118) Ebd., Pos. 428–31.
  • (119) Ebd., Pos. 582–95.

Kapitel 5

  • (120) C. Manisha Chandalia et al., »Beneficial Effects of High Dietary Fiber Intake in Patients with Type 2 Diabetes Mellitus«, New England Journal of Medicine, 342 (2000): 1392–98, DOI: 10.1056/NEJM200005113421903.
  • (121) M. Wien et al., »A Randomized 3×3 Crossover Study to Evaluate the Effect of Hass Avocado Intake on Post-ingestive Satiety, Glucose and Insulin Levels, and Subsequent Energy Intake in Overweight Adults«, Nutrition Journal, 12 (2013): 155, DOI: 10.1186/1475-2891-12-155.
  • (122) »Potassium«, University of Maryland Medical Center, http://umm.edu/health/medical/altmed/supplement/potassium, aufgerufen am 14. August 2017.
  • (123) M. E. Cogswell et al., »Sodium and Potassium Intakes among U.S. Adults: NHANES 2003–2008«, The American Journal of Clinical Nutrition, 96, Nr. 3 (2012): 647–57, DOI: 10.3945/ajcn.112.034413.
  • (124) M. L. Dreher und A. J. Davenport, »Hass Avocado Composition and Potential Health Effects«, Critical Reviews in Food Science and Nutrition, 53, Nr. 7 (2013): 738–50, DOI: 10.1080/10408398.2011.556759.
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Kapitel 6

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Kapitel 7

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Kapitel 8

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Kapitel 9

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Kapitel 10

  • (152) »Overweight and Obesity Statistics«, U. S. Department of Health and Human Services, https://www.niddk.nih.gov/health-information/health-statistics/Pages/overweight-obesity-statistics.aspx, aufgerufen am 14. August 2017.
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Kapitel 11

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  • (170) D. Z. Kochan et al., »Circadian Disruption and Breast Cancer: An Epigenetic Link?«, Oncotarget, 6, Nr. 19 (2015): 16866–82. DOI: 10.18632/oncotarget.4343.
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  • (172) D. Peretti et al., »RBM3 Mediates Structural Plasticity and Protective Effects of Cooling in Neurodegeneration«, Nature, 518, Nr. 7538 (2015): 236–39, DOI: 10.1038/nature14142.

Anhang A

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  • (221) »Non-Alcoholic Fatty Liver Disease«, American Liver Foundation, http://www.liverfoundation.org/abouttheliver/info/nafld/, aufgerufen am 15. August 2017.
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